Can plastic chemical affect ovarian function?

Eating canned foods during pregnancy can expose your baby to the industrial chemical bisphenol A (BPA), which can later affect their reproductive health, warns a study. Detectable in surface water and soil sediments, BPA is used in many industrial processes (epoxy resins, polycarbonate plastic production) and in the lining of food cans. While the greatest exposure is dietary from canned foods and plastic containers, skin exposure is a secondary route of smaller exposure.

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To examine whether a prenatal environmental exposure can pose a real threat to human ovarian function, researchers examined existing literature from 2000 to June 2018.

"We found there is mounting evidence for the effects of these exposures in the prenatal period, a particularly vulnerable time of development," explained an Assistant Professor. 

"Whether there are causative associations with human ovulation disorders needs to be further studied."

BPA has been known to disrupt the endocrine system in fetuses and children (as well as adults), causing long-term health effects as well as in reproductive health outcomes. According to the researchers, ovarian development and function represent complex coordination of processes, starting early during prenatal development. Early aberrations have the potential to carry through the female reproductive lifespan.

The researchers believe their findings are particularly relevant since there are implications for both foetal and women's health. 

"Understanding the effect that BPA exposure has on ovarian outcomes may contribute to the treatment approach taken for diseases and disorders in which ovarian dysfunction is a manifestation such as infertility, polycystic ovary syndrome and premature ovarian failure," the Asst. Prof. added.
The researchers hope their study raises awareness of the lasting effects that harmful prenatal exposures may have and that additional studies looking at the long-term effects of endocrine disrupting chemicals in diverse populations will be undertaken.

Maternal exposure to plastic chemical during pregnancy may up asthma risk in kids

A mother’s exposure to chemicals used to soften and increase the flexibility of plastic, during pregnancy and breastfeeding may increase the risk of children developing allergic asthma, says a study.

The chemical – called phthalates – can enter our bodies through the skin, foodstuffs or respiration. It has also been well-known to affect the hormone system and can thereby have an adverse effect on our metabolism or fertility.

“The results of our study demonstrate that phthalates also interfere with the immune system and can significantly increase the risk of developing allergies,” said Tobias Polte, environmental immunologist at Helmholtz University in Germany. 

For the study, appearing in the Journal of Allergy and Clinical Immunology, the team examined the urine of pregnant women and their influence on the newborn-allergy-risk. 

“There was a clearly discernible relationship between higher concentrations of the metabolite of benzylbutylphthalate (BBP) in the mother’s urine and the presence of allergic asthma in their children,” added Irina Lehmann from Helmholtz Centre for Environmental Research. 

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Common plastic chemical may increase breast cancer risk

An endocrine-disrupting chemical commonly found in polycarbonate hard plastics, currency bills and thermal paper receipts may potentially interfere with the body's hormones to increase the aggressiveness of breast cancer, a new study has showed.

Bisphenol S (BPS), a substitute for the chemical bisphenol A (BPA) in the plastic industry, acts like estrogen in multiplying breast cancer cells.

Most breast cancers are estrogen receptor positive, and, according to the National Cancer Institute, 55 to 65 per cent of women who inherit a harmful mutation in the BRCA1 gene -- will develop breast cancer.

"If a woman has a mutated BRAC1 gene and uses products containing BPS, her risk for developing breast cancer may increase further," said principal investigator, Sumi Dinda, Associate Professor at Oakland University in Michigan.

"Despite hopes for a safer alternative to BPA, studies have shown BPS to exhibit similar estrogen-mimicking behaviour to BPA," Dinda added.

The results were presented at ENDO 2017, the Endocrine Society's 99th annual meeting in Orlando.

For the study, the team used two commercially available breast cancer cell lines obtained from women with estrogen-receptor-positive breast cancer, to expose the cancer cells to varying strengths of BPS or to an inactive substance as a control.

Compared with the control, BPS heightened the protein expression in estrogen receptor and BRCA1 after 24 hours, as did estrogen.

After a six-day treatment with BPS, the breast cancer cells in both cell lines reportedly increased in number by 12 per cent at the lowest dose (4 micromolars) and by 60 per cent at eight micromolars.

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An endocrine-disrupting chemical commonly found in polycarbonate hard plastics, currency bills and thermal paper receipts may potentially interfere with the body's hormones to increase the aggressiveness of breast cancer, a new study has showed.

Bisphenol S (BPS), a substitute for the chemical bisphenol A (BPA) in the plastic industry, acts like estrogen in multiplying breast cancer cells.

Most breast cancers are estrogen receptor positive, and, according to the Nationa ..

Read more at:
http://economictimes.indiatimes.com/articleshow/57985562.cms?utm_source=contentofinterest&utm_medium=text&utm_campaign=cppstAn endocrine-disrupting chemical commonly found in polycarbonate hard plastics, currency bills and thermal paper receipts may potentially interfere with the body's hormones to increase the aggressiveness of breast cancer, a new study has showed.

Bisphenol S (BPS), a substitute for the chemical bisphenol A (BPA) in the plastic industry, acts like estrogen in multiplying breast cancer cells.

Most breast cancers are estrogen receptor positive, and, according to the National Cancer Institute, 55 to 65 per cent of women who inherit a harmful mutation in the BRCA1 gene -- will develop breast cancer.

"If a woman has a mutated BRAC1 gene and uses products containing BPS, her risk for developing breast cancer may increase further," said principal investigator, Sumi Dinda, Associate Professor at Oakland University in Michigan.

"Despite hopes for a safer alternative to BPA, studies have shown BPS to exhibit similar estrogen-mimicking behaviour to BPA," Dinda added.

The results were presented at ENDO 2017, the Endocrine Society's 99th annual meeting in Orlando.

For the study, the team used two commercially available breast cancer cell lines obtained from women with estrogen-receptor-positive breast cancer, to expose the cancer cells to varying strengths of BPS or to an inactive substance as a control.

Compared with the control, BPS heightened the protein expression in estrogen receptor and BRCA1 after 24 hours, as did estrogen.

After a six-day treatment with BPS, the breast cancer cells in both cell lines reportedly increased in number by 12 per cent at the lowest dose (4 micromolars) and by 60 per cent at eight micromolars.