Sunday, August 30, 2026

The real reasons you procrastinate – and how to stop, according to science

Faced with a task or project, do you repeatedly tell yourself that you’ll do it “tomorrow”, “soon”, or “after watching just a few more videos”?

If so, as someone who has studied the science of procrastination for years, I can reassure you that you are not alone. Around 20% of adults procrastinate regularly.

Those of us who do this – myself included – may also experience a range of issues and outcomes that research shows are linked to procrastination, including worse exam grades, lower income, lack of sleep, and poor mental and physical health.

Procrastination can be painful and frustratingly difficult to deal with. A key reason is that a lot of what we hear about this common behaviour is wrong.

Perhaps the biggest misconception is that it comes from not caring enough, or from being lazy. By this false reading, people who procrastinate just need to “try harder”. Such misperceptions can lead to feelings of guilt and shame.

In reality, procrastinators usually intend to work every bit as hard as their peers – if not harder. But a defining feature of procrastination is the gap between intentions and actions. Not only does “just trying harder” not work, it often backfires by sidetracking us from solutions that do.

What really causes procrastination?

Essentially, our brains are hardwired to prioritise what will make us feel better right now, over what will make us feel better in the long term and overall.

So, when we worry that doing something might be painful or difficult, our brain nudges us to put it off as a defence mechanism, to postpone the pain we expect it to bring.

This can happen when we feel that studying will be frustrating, or an upcoming exam will be stressful. Or, we may be worried that our writing or presentation won’t be perfect.

Perfectionism can be especially problematic when we worry our work will be criticised by others – whether or not that’s likely to happen in reality.

Our brains can be further confused when we’re faced with tempting alternatives that we know will be immediately gratifying, such as social media feeds that are hyperoptimised to capture our attention for as long as possible.

Additional issues can make it even harder for us to course-correct our brains when procrastinating about a deadline. A key one is being tired – or worse, burned out – from working too hard for too long on tasks that are draining.

This can then end up in a vicious cycle. We are too exhausted to do what we need to, so we procrastinate on it, which stresses us out and drains us further, tightening procrastination’s grip.

Studies have also linked procrastination with ADHD, a neurodevelopmental disorder that can make it harder for people to stick to one particular task.

Fortunately, just as misunderstanding procrastination can set us on the wrong path for overcoming it, understanding the real reasons can help us figure out how to stop.

How to stop procrastinating

There are lots of reasons for procrastinating, each of which may call for a different solution. Start by asking yourself why you are procrastinating this time? (It may not always be for the same reason.)

Are you exhausted? Are you getting distracted by your phone? Are you afraid you’ll make mistakes? Or is it some other issue, or combination of issues, that’s getting in your way of achieving the goal or task you’ve been set, or set yourself?

If you’re unsure, try comparing cases where you took action with ones where you put it off endlessly. What was the difference?

Alternatively, pretend that a friend is in a similar situation and reflect on their problem instead. Adding this psychological distance could help you see things more clearly.

Next, try asking what one change you can make right now that will make you more likely to complete the task. Some are easier than others, admittedly.

If you’re getting distracted by your phone, for example, turn it off before you start the task. If you’re working in a library or cafe, put away your phone inside your bag when you sit down.

If fear of a particular outcome is the problem, that’s harder. But try asking yourself: how likely is it, really, that whatever you’re most afraid of will actually happen? Often it’s a lot less likely, or less scary, than you think.

Another strategy is to work through this worst-case scenario, considering how you could lessen the negative impact if it does happen.

I discuss other causes of procrastination, and more solutions to them, in my new book, Solving Procrastination: The Science of Why We Put Things Off and How to (Finally!) Stop. Briefly, these include suggestions such as:

  • Unpack unclear tasks into manageable micro-steps, to make your path forward feel clear and attainable.

  • Build your self-belief by identifying and achieving consistent wins, to silence the inner pessimist that’s holding you back.

  • Structure your schedule according to your productivity rhythms, to work with your natural preferences, not against them.

Above all, overcoming procrastination is never simply about “trying harder”. Rather, it’s about figuring out the real reasons you tend to put things off – and then addressing them using the techniques that are right for you.

 

This is only for your information, kindly take the advice of your doctor for food, medicines, exercises and so on.   

 

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5 signs you could be intolerant to caffeine

In 2026, there’s almost nothing we don’t know about our bodies. From how we sleep to how the food we eat interacts with our microbiome to exactly what our blood sugar gets up to throughout the day, rare is the metric that remains a mystery. And I recently decided to take things a step further: I completed the 10X Health System  genetic test, to learn what secrets were lurking in my genes and how I could positively harness the information for the benefit of my health.

Besides having a slightly dubious detoxification system, the main takeaway from my results was my body’s genetic inability to process caffeine. I’d long suspected I had a systemic intolerance to coffee, matcha and caffeinated tea, largely because of how off-kilter it makes me feel.

You may not want to know whether or not your body is particularly adept at processing caffeine and I respect that – so long as you’re not pounding a dozen espressos a day, you should be fine ( current guidelines suggest adults should not exceed 400mg of caffeine a day, which is the equivalent of four to five cups of coffee or matcha). If you are interested, though, read on.

How your genes determine your caffeine intolerance

Prepare yourself for some unpronounceable genetic terms. “The CYP1A2 enzyme gene in the liver is responsible for breaking down roughly 95 percent of all caffeine consumed,” Dr Pascale Ricci, head of precision nutrition at Reviv Global, a brand that works alongside 10X Health, tells me. “The rs762551 SNP (single-nucleotide polymorphism) determines how fast or slow it occurs.”

Right, let’s break that down: rs762551 is a variation of the CYP1A2 enzyme gene. It’s what determines how quickly or slowly your body can break down caffeine. Committing either term to memory is not important, but understanding the role they play in your caffeine tolerance is. A genetic test, like the one I did, produces one of three results: you’re either an AA, AC or CC.

AA: the fast metaboliser

Approximately 41 percent of people are AA or fast metabolisers of caffeine, meaning their CYP1A2 enzyme works efficiently. “Caffeine is cleared relatively quickly, with a typical half-life of around two and a half to four and a half hours,” Ricci explains. If you know someone who can have a fresh cup of coffee at 6pm and still sleep soundly, chances are they’re an AA.

AC: the intermediate metaboliser

Join me in the middling club, along with around 43 percent of the population! An AC gene variant suggests that one of your parents gave you a functioning gene and one gave you a defective one. This makes us okay at metabolising caffeine: not brilliant, not terrible.

CC: the slow metaboliser

Finally, 16 percent of the population are estimated to be slow caffeine metabolisers, with two copies of the slower gene variant. “This means caffeine lingers in the bloodstream for significantly longer and leads to higher plasma caffeine levels after a cup of coffee,” when compared to someone with an AA variant, Dr Ricci explains.

“In simple terms, if two people drink the same latte, the slow metaboliser (CC) will have caffeine circulating in their body for much longer than the fast metaboliser (AA), meaning the  effects (both the good and the bad) last longer and feel stronger.”

How to tell which type of caffeine metaboliser you are

Short of a genetic test, Dr Ricci has a few pointers of things to look out for when thinking about how well you metabolise caffeine.

Sleep disturbances

Difficulty falling asleep and poor sleep quality, even when keeping caffeine consumption to the morning, can indicate slow caffeine metabolisation.

 

Mood changes

“Anxiety, jitteriness or nervousness, particularly when consuming over 200mg of caffeine,” is another sign slow metabolisers may notice, she says. A prolonged “wired” feeling is another telltale sign.

Heart

A pounding heartbeat or palpitations can be an indication of a caffeine reaction, though they can also be a sign of other, more worrying health concerns. Contact the emergency services or your GP if a new symptom arises, persists or becomes concerning.

Digestion

An upset stomach, acid reflux or diarrhoea can all be signs of caffeine intolerance, though they may also have other causes.

Genetic test

For a definite answer, a genetic test is the way to go. It proved my suspicions: thanks to one of my parents, an AC gene variation meant my body struggled to break down caffeine and, according to my report, is best avoided.

How to enjoy caffeine regardless of results

Less, not none

Dr Ricci suggests curbing your daily caffeine intake to less than 200mg a day, which is the equivalent of one cup of coffee. A double-shot of coffee contains anywhere between 120mg and 160mg of caffeine, while a single shot would sit in the 60mg to 80mg range.

Timing matters

“Front load caffeine by drinking it in the morning,” Dr Ricci recommends. “Caffeine lingers for longer in slow metabolisers and consuming it later in the day is more likely to disrupt sleep.” Find a cut-off point that works for your lifestyle and make it a hard stop. That could be after your first cup or go by the clock and avoid it altogether after midday.

Go low

If you want to drastically cut down but can’t abandon the habit entirely, green tea and half-caffeinated coffee are good options. Green tea contains around 30mg to 50mg of caffeine per cup, which, when swapping from a double-shot habit, is a marked reduction.

Make it a decaf chaser

A good rule of thumb is to make every follow-up drink (whatever you reach for after you’ve had your one or two cups of coffee) a decaffeinated one.

Be aware of the bigger picture

Dr Ricci warns that some common medications (“antibiotics, certain cardiovascular drugs, oral contraceptives and some anti-depressants”) slow down the CYP1A2 enzyme gene activity, which can compound the already lingering effects of caffeine. “You may need to reduce the amount of caffeine you drink further when starting new medication,” she cautions. The best approach is to speak with your doctor when starting any new medication or supplement.

Taper, don’t cold turkey

“Tapering off caffeine, rather than quitting abruptly, tends to reduce overall consumption,” Dr Ricci finishes. “It also helps to minimise withdrawal symptoms like headaches and fatigue.”

 

 

 

This is only for your information, kindly take the advice of your doctor for food, medicines, exercises and so on.   

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